We apply the Displacement Framework to Type 2 Diabetes (T2D), treating normal insulin sensitivity and glucose homeostasis as S0_metabolic. T2D develops through progressive metabolic displacement, with visceral adiposity as a displacement amplifier. Insulin resistance is formalized as wrong-attractor capture via four self-reinforcing loops. HbA1c is recast as a displacement-integral proxy.
Complications are secondary wrong-attractor captures. Unusually among chronic diseases, T2D is often reversible: DiRECT trial data, bariatric surgery, and intensive lifestyle intervention are analyzed as return-path interventions. T2D and T1D are distinguished as wrong-attractor capture versus return-path destruction.
Phronesis